EEG Section, National Institute of Neurological Disorders and Stroke (NINDS), National Institutes of Health (NIH), Bethesda, MD, HSRDP, Portland VA Medical Center, Portland, OR, USA
- Key words:
CBZ, carbamazepine, CNS, central nervous system, FPHT, osphenytoin, HSV, Herpes simplex virus, JEV, Japanese encephalitis virus, NCSE, non-convulsive status epilepticus, PHT, phenytoin, PLEDS, periodic lateralized epileptiform discharges, PCR, polymerase chain reaction, PRIS, propofol infusion syndrome, SE, status epilepticus, SLE, St Louis encephalitis, VPA, valproic acid, WNV, West Nile virus
- DOI : 10.1684/epd.2007.0056
- Page(s) : 98-103
- Published in: 2007
West Nile virus (WNV) has re-emerged with a much wider geographic distribution and a higher incidence than ever. In spite of some recent reports on the neurological manifestations and EEG changes caused by WNV encephalitis, there are few data on the incidence of seizures, status epilepticus or post-encephalitic epilepsy. There is also no systematic review of EEG changes caused by WNV encephalitis that is based on a large series of patients. Here, we review the pertinent literature, and report the electroclinical evolution and therapeutic complexity of a patient with WNV encephalitis who developed refractory, non-convulsive status epilepticus.