John Libbey Eurotext

The role of the temporal pole in the genesis of temporal lobe seizures Volume 4, supplément 1, Supplement 1, September 2002


   
   Figure 1. Mesio-temporal lobe epilepsy syndrome: a suggestive history, a specific anatomical context, relatively stereotyped seizures, poor prognosis.



   
   Figure 2. SEEG traces during temporal lobe seizures. A. Fast activity involving both hippocampus and amygdala more or less simultaneously; temporal pole changes are clearly transmitted. B. Low voltage fast activity simultaneously involving the amygdalo-hippocampal complex and the temporal pole.



   
   Figure 3. Interictal SEEG trace from a patient with hippocampal sclerosis. Continuous, rhythmic spikes on the internal side of the temporal pole (Tpi), seen in the form of lateral temporal polar (Tp) spike and waves and discernible in the hippocampus (Hc) but not in the amygdaloid nucleus (AN); note the rhythmic slow waves in the temporal neocortex (aT2, pT2 and aT1).



   
  

Figure 4. SEEG trace of the discharge during a paucisymptomatic seizure in a patient with hippocampal sclerosis. The low-voltage fast activity remains confined to the temporopolar region, with more activity on the lateral (eTP) than on the mesial side (iTP). The patient sometimes reported concomitant tingling at the tip of her nose.