JLE

Epileptic Disorders

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Ocular compression a century on: time for a thumbs-off approach? Volume 10, numéro 2, June 2008

Figure 1 Three consecutive panels of EEG/ECG recording to show AES induced by OC. Each strip lasts 20 s. In the upper panel OC takes place between the two vertical lines. Asystole begins after only two QRS complexes on the bottom ECG channel. After 12 s the EEG is isoelectric (with rhythmic artefacts from down-beat nystagmus in channels FP2-T4 and FP1-T3). Tonic EMG on the ECG channel reflects the anoxic seizure. Midway across the centre panel and while the EEG is still isoelectric rhythmic 2-3/s. jerk artefacts are seen on the EEG and ECG channels. By the start of the lower panel rhythmic EEG spike and wave is visible, confirming the epileptic component of the AES. Clonic jerking only lasted 28 s but the child was unresponsive for a further 8 minutes (modified from Stephenson [1990], and reproduced with permission of Mac Keith Press).