John Libbey Eurotext



Etiology of the sugar beet rhizomania. Volume 11, issue 6, novembre - décembre 2007


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Institut de biologie moléculaire des plantes, Laboratoire propre du CNRS (UPR 2357) conventionné avec l’Université Louis-Pasteur (Strasbourg 1), 12, rue du Général-Zimmer, 67084 Strasbourg, INRA, 28, rue de Herrlisheim, 680021 Colmar, Università di Bologna, Dipartimento di Scienze e Tecnologie Agroambientali, area Patologia Vegetale, Viale G, Fanin, 40 - II piano, 40127 Bologna, Italie, Unité de phytopathologie, UCL, Croix du Sud 2 bte 3, 1348 Louvain-la-Neuve, Belgique

Beet necrotic yellow vein virus is responsible for sugar beet rhizomania. Root proliferation is characteristic of the viral infection and lead to sugar losses. Pathogenicity is particularly linked to the expression of RNA-3-encoded p25. The extensive use of viral tolerant crops allows maintenance of sugar yields but also permits viruliferous vector to be maintained and therefore the appearance of resistance breaking isolates. The resistance breaking isolates present some amino acid variations within the p25 protein sequence, a key determinant in BNYVV pathogenicity. Here, we will review the molecular biology of BNYVV, of its vector and the antiviral strategies that may be used against rhizomania.